Avaliação colostral e de níveis séricos imunes de bezerros holandezes oriundos de vacas acometidas ou não por mastite

• Ano de defesa: 2013
• Autor(a) principal: Santos, Guilherme Gonçalves Fabretti [UNESP]
• Orientador(a): Feitosa, Francisco Leydson Formiga
• Tipo de documento: Dissertação
• Tipo de acesso: Acesso aberto
• Idioma: por
• Instituição de defesa: Universidade Estadual Paulista (Unesp)
• Palavras-chave em Português: Mastite; Colostro; Bezerro; Citocinas; Imunoglobulinas; Proteínas; Imunidade celular; Immune passive transfer
• Link de acesso: http://hdl.handle.net/11449/88173

The aim of this study was to evaluate the transfer of passive immunity (TPI) in Holstein calves delivery from cows with subclinical and clinical mastitis. Four groups of studies were evaluated. Group I (GI) 10 healthy primiparous cows with SCC ≤ 300,000 cells/mL, Group II (GII) 10 healthy multiparous cows with SCC 301-600,000 cells/mL, Group III (GIII) 10 multiparous cows with mastitis records that calved with subclinical mastitis with SCC> 600,000 cells/mL with normality in udder and colostrum and Group IV (GIV) 10 multiparous cows with mastitis records that calved with clinical mastitis with SCC> 600 000 cells/mL with changes in the udder and/or colostrum. Blood samples and colostrum were collected immediately after birth, 24 and 48 hours after delivery/birth. The microbiological culture showed that Streptococcus sp. and Staphylococcus sp. were the most common bacteria isolated from cows with subclinical and clinical mastitis respectively. The SCC stipulated for the control group cannot be followed, since the secretions of healthy animals showed average values greater than 992,000 cells/mL of colostrum. CMT can be useful to confirm mastitis in post-partum period. The ultrasound was effective in determining parenchyma affected by infection. No differences were observed between the values of the protein constituents of colostrum and values of cytokines in multiparous and primiparous cows. Ingestion of colostrum derived from cows with clinical and subclinical mastitis is unlikely to be an important contributor to failure of passive transfer in calves